DOT1L cooperates with the c-Myc-p300 complex to epigenetically derepress CDH1 transcription factors in breast cancer progression
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Summary #
Results #
DOT1L induces breast tumorigenesis by enhancing CSC activity #
DOT1L expands CSCs via increasing EMT-TFs #
DOT1L epigenetically derepresses EMT-TFs with c-Myc and p300 #
c-Myc is prerequisite for DOT1L-induced EMT-TF regulation #
Discussion #
Methods #
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